![]() They penetrate the skin more deeply than UVB rays, affecting the DNA of the cells in the dermis, attacking cell membranes, and changing the proteins that make up collagen and elastin, which support the skin's fibrous structure. Ultraviolet A (UVA) rays - longer than UVB rays - can also do lasting damage. More seriously, it contributes to the development of melanoma and other skin cancers. ![]() This kind of genetic damage causes both freckling and the mottled brown of age spots. The melanocytes react by stepping up production of melanin and sending melanasomes to the skin's surface to act as a filter against the sun's rays actually damage the DNA of the pigment cells. Finally, UVB radiation attacks the skin's melanocytes (pigment cells). Other UVB rays can affect the immune system and interfere with the skin's ability to repair itself. These chemicals irritate the tiny blood vessels in the dermis, which swell and create the surface redness of the burn.Īt the same time, the UVB rays affect the genetic material of the epidermis, which causes the damage that may lead to skin cancer. When you burn, the skin responds to UVB rays by producing chemicals called inflammatory mediators, some of which seep down into the dermis, the skin's middle layer. ![]() Ultraviolet B (UVB) rays are short, high-energy wavelengths that are absorbed by the epidermis, the outermost layer of the skin. The sun gives off invisible rays of ultraviolet light. Tanned skin will forever contain cells whose genetic structures have been permanently damaged by the sun. There is no such thing as a healthy tan, according to dermatologists, who look at a tan and see a sign of injury.
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